Low Testosterone Levels Post Cycle
Unfortunately, this is easier said than done, because the levels of various hormones and other substances that were circulating around your body during the cycle huge amounts of testosterone, insulin-like growth factor, growth hormone, and lower amounts of muscle-wasting glucocorticoids are now changing.
Sadly, they are making way for lower amounts of the hormones we want for building muscle, and higher amounts of the catabolic ones. What needs to be done, as quickly as possible, is to get your body to begin production of your own natural anabolic hormones, and produce less of the catabolic ones.
Unfortunately, your body has other plans. Is all of this necessary? No, not at all. You can skip to the end of the article and look for a little chart I made — the extent of my computer skill — which has all of the dosage recommendations and compounds involved to properly recover from your cycle. Remember, during this lag-time you lose gains, so we really need to make it as short as possible.
First, we need to understand a bit of what is going on in your body, what causes it to happen naturally, and what hormones are performing what function. At the age of , Gonadatropin Releasing Hormone GnRH is increasingly released from the Hypothalamus, in turn causing the secretion of Follicle Stimulating Hormone FSH and Luetenizing Hormone LH from the pituitary, and finally the male gonads testes are then stimulated by those pituitary hormones LH and FSH.
What does this mean? In the case of testosterone, for example, one of the messages it sends to the cell is to increase nitrogen retention in your body, thus allowing you to use more of the protein you take in, and build more muscle. In the case of testosterone or anabolic steroids in generalthis transcription causes a lot of different anabolic effects to take place: an increase in IGFa decrease in cortisol, an increase in Red Blood Cell count, and the increased protein synthesis I already told you about.
This is not to say that AR binding is the only thing that causes anabolic or androgenic effects, however. Oxymetholone and Methandrostenolone Anadrol and Dianabol both bind very weakly to the AR yet are both highly anabolic and androgenic. This can be seen in when males grow body hair experience voice changes, as experience genital development and growth.
Lets review one of the first things I mentioned, ok? Ok, now, once testosterone is created however, it has the ability to in turn to undergo various metabolic processes that will inhibit GnRH, which in turn inhibits the secretion of LH and FSH, and that brings a halt to natural testosterone production. Once testosterone has stopped being produced, it no longer sends this negative signal, and GnRH eventually begins to do its job again.
This negative feedback loop is partially why we use anabolic steroids…we want more testosterone for anabolic purposes or more Anavar or whatever than our body will let us produce not that our bodies produce Anavar, but you get the idea. Am I being repetitive? Remember, the negative feedback loop is, of course, no problem while we are on a cycle. Want more testosterone or androgens in your body?
Fill up a few more syringes! But all good things come to an end, and most of us choose to end our cycles at some point. SO what we need to do is coax the body into quickly producing its own androgens. Nolvadex Tamoxifen is a SERM Selective Estrogen Receptor Modulator, which means that it has the ability to act as an anti-estrogen with regard to certain genes, yet also acting as an estrogen with respect to others.
However, it acts as an anti-estrogen in the pituitary, thus increasing LH and FSH, which results in an increase in testosterone. Nolvadex does this by actually competing for the receptor site in breast tissue, and binding to it.
Testosterone: What’s a Normal Testosterone Level in Men? | The Art of Manliness
Nolvadex also has some important features for the steroid using athlete.
Well, basically because it takes much more to do the same thing. This most likely indicates some kind of upregulation of the LH-receptors due to the anti-estrogenic effect Nolvadex has at the pituitary. Although both Nolvadex and Clomid are both SERMs, they are actually quite different. SO how much Nolvadex should you use during PCT? I favor using www.urbanmessenger.org, although to be totally honest, you can probably even get away with far less than that.
So that effectively suggests Nolvadex can not be used at Mega-doses to get a mega-increase in your natural hormones. Arimidex an Aromatase Inhibitor —which means it stops the conversion of testosterone into estrogen-another drug used to fight breast cancer like Nolvadex exhibits basically the same effects when.
Post Cycle Therapy | Steroids Cycles
It must be, therefore, that your body can only respond with so much vigor to any one drug in those families.
So lets add in another drug or two, ok? This way we can use reasonable doses of a few drugs and produce some synergy…hopefully decreasing our recovery time. Obviously, as you can guess from the name, it is a substance that stimulates the gonads hence: gonadotropin. It does this by initiating gene transcription that is identical to that of Luetenizing Hormone, thereby causing the Leydig Cells to produce testosterone.
We can stimulate LH and FSH production with our Nolvadex, and then directly stimulate the Leydig Cells as well, to produce tons of testosterone by different routes! This LH downregulation can cause an increase in steroienic cholesterol the cholesterol earmarked by your body for conversion into testosterone. Thus, after the initial HCG induced surge in testosterone is over, if you have used enough to downregulate your LH-receptors and increase estrogen too much, then more steroienic cholesterol is available.
This is telling me that less is being converted to testosterone. Low LH post-cycle is not the primary cause of slow recovery, because LH generally rises to levels above baseline after a cycle much sooner than testosterone production does. This is probably because the pituitary is working very hard to get your atrophied Leydig cells to start producing testosterone again.
This is great news for us, because we are going to be using HCG during PCT, when we need to get back some HPTA function, and not when we have levels of gonadatropins high enough to cause HCG-induced desensitization.
But are we still risking some inhibition and possibly delaying our recovery by using HCG? Another factor involved is the induction of testicular aromatase, which raises estrogen levels, again causing inhibition. Unfortunately, yet another process, the downregulation of the Leydig Cell LH receptor itself, seems to also play a role in high dose HCG testicular desensitization. Lets add in an Aromatase Inhibitor! With this PCT, there will be a rapid increase in LH, FSH, and testosterone, as well as almost a complete block on all the factors that could be causing your natural hormones to be delayed in returning to baseline.
For this reason, I feel that the second your cycle is over is when you should start this PCT a week after your last shot, or the day after your last pill is fine. And how long do we run this for? Well…we need to stop the HCG relatively soon for reasons discussed earlier. But the Nolvadex, and Aromasin can be used for awhile longer. And with no further delays, here are my recommendations for PCT: Filed Under: Steroid Articles Tagged With: PCTpost cycle therapy. Will this inhibit the benefits of hcg during my pct?
Should I stop using hcg until I need itfor pct? Do you think I should taper down or stop cold turkey test and deck? What should I do to remain my gain? You must be logged in to post a comment. DeutschEnglish. Aromasin Exemestane is one of those weird compounds that nobody really knows what to do with. What we generally hear. But then, so do I…. Under the control of this heightened state of androgens, you also go through androgenic development as well as anabolic development.
The chart below clearly shows this process, displaying both the negative and positive feedback system s :. This suggests that a pre-exposure to normal LH levels or gonadatropins in general is necessary for HCG-induced Leydig Cell desensitization.
Lets solve that pesky estrogen problem now…. And with no further delays, here are my recommendations for PCT:. Breast Cancer Res Treat.
Disparate effect of clomiphene and tamoxifen on pituitary gonadotropin release in vitro. Adashi EY, Hsueh AJ, Bambino TH, Yen SS. Effect of lower versus higher doses of tamoxifen on pituitary-gonadal function and sperm indices in oligozoospermic men.
J Clin Endocrinol Metab. Hypogonadism Postgrad Med J. Mol Cell Endocr inol. Proc Natl Acad Sci U S A. Kinetics of the steroienic response of the testis to stimulation by hCG. Forest MG, Roulier R.
Effect of vitamin E on function of pituitary-gonadal axis in male rats and human subjects. Umeda F, Kato K, Muta K, Ibayashi H. J Steroid Biochem Mol Biol. Inhibitory effect of combined treatment with the aromatase inhibitor exemestane and tamoxifen on DMBA-induced mammary tumors in rats. Bruning PF, Bronfer JMG, Hart AAM, Jong-Bakker M, tamoxifen, serum lipoproteins and cardiovascular risk, Br. Stimulation of calcitonin secretory capacity by increased serum levels of testosterone in men treated with tamoxifen.
Hormonal changes in tamoxifen treated men with idiopathic oligozoospermia Exp Clin Endocrinol. My supplier recommended the use of hcg about mid — way through my cycle because my testicles were literally gone. Thanks Log in to Reply. Log in to Reply.
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Read previous post: Rationale for the Use of Aromasin with Tamoxifen During Post Cycle Therapy Aromasin Exemestane is one of those weird compounds that nobody really knows what to do with.